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The possible mechanistic model anticipates that, in the absence of a stimulus, GSTP1-1 averts IκBα degradation and GSTP1-1-mediated S-glutathionylation shuts down IKK activity, providing a versatile mechanism by which GSTP1-1 represses NF-κB activation
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caldus A Southern blot analysis was performed, as described earlier (Wen et al., gr mutant and wild type of A
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