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Trimethylamine N-oxide promotes atherosclerosis via CD36-dependent MAPK/JNK pathway

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While increased intraocular pressure (IOP) has long been regarded as the principal risk factor, accumulating evidence demonstrates that lowering IOP alone cannot fully prevent disease progression, especially in normal-tension glaucoma (NTG), highlighting the important roles of optic nerve perfusion abnormalities, vascular autoregulatory dysfunction, and disturbances in neural metabolism in the pathogenic process (25)