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Soluble Fas ligand (sFASL): Soluble Fas ligand (sFASL) is the circulating form of membrane-bound Fas ligand, a member of the TNF superfamily, and plays a critical role in regulating apoptosis and immune responses

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[29] Mouse models with this mutation do not exhibit the classical mitochondrial vacuolation pathology seen in G93A and G37R ALS mice and unlike G93A mice, deficiency of the major mitochondrial antioxidant enzyme, SOD2, has no effect on their disease course