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Theoretical rationale for combination approaches exists based on the complementary mechanisms of telomerase activation (preventing new senescent cells) and senolytic therapy (eliminating existing senescent cells), but this remains entirely speculative without experimental data

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When desensitized, the GPCR either persists at the cell surface but becomes refractory to repeated stimulus or, in some instances, the GPCR is degraded and is restored later by protein synthesis and protein processing