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As mentioned above, tumor cells are more susceptible to electrical charges than normal cells, thus allowing more drugs to enter the tumor cells, enabling passive targeting functions and accumulation of more drugs at the tumor site

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For example, disruption of ERK1/2 signaling enhances both the development and expression of morphine tolerance with microinjections of the MEK inhibitor U0126 [1,4-diamino-2,3-dicyano-1,4-bis(methylthio)butadiene] into the periaqueductal gray area (Macey et al., 2009)