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This PDE3B-specific model of Cu-dependent regulation of enzyme activity in lipolysis may well be general, as Cu is known to regulate the activity of a number of protein kinases involved in various cell signaling pathways (Figure 5A)

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the D-Nle ⁇ Trp- Ile-NH-(CH 2 )5-CONH 2 analog which exhibits intermediate inhibition of dimerization, predictably has a moderate affinity for HGF and a moderate ability to inhibit HGF-dependent processes ( Figures 25, 26, 27)