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high glutathione circadian rhythm An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in Lutein
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high glutathione circadian rhythm An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in Lutein
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high glutathione circadian rhythm An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in Lutein
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high glutathione circadian rhythm An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in Lutein
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Using bioinformatics analysis, real-time quantitative polymerase chain reaction (qPCR), and co-immunoprecipitation, assays, Yao et al
high glutathione circadian rhythm An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in Lutein
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